Anti-FBLN5PolyclonalAntibody-一抗-抗體-蛋白與免疫
2023-10-29
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Anti-FBLN5 Polyclonal Antibody品牌：solarbio | 貨號：K006131P| 英文名稱 | Anti-FBLN5 Polyclonal Antibody |
|---|---|
| 宿主 | Rabbit |
| 別名 | ADCL2;ARCL1A;ARMD3;DANCE;EVEC;FIBL-5;HNARMD;UP50 |
| 應用 | WB |
| 稀釋比例 | WB 1:500-2000. |
| 交叉反應 | Human Mouse |
| 蛋白分子量 | 50kDa |
| Gene ID | 10516 |
| 保存 | Store at -20°C. Avoid freeze / thaw cycles. |
| 儲存液 | Buffer: PBS with 0.02% sodium azide, 50% glycerol, pH7.3. |
| 純化方法 | Affinity purification |
| 亞型 | IgG |
| 免疫原 | Recombinant protein of human FBLN5 |
| 性狀 | 液體 |
| Public Immunogen Range | Recombinant protein of human FBLN5 |
| Subcellular Locations | Secreted extracellular matrix extracellular space |
| Swiss Prot | Q9UBX5 |
| 克隆類型 | Polyclonal Antibody |
| 背景資料 | The protein encoded by this gene is a secreted, extracellular matrix protein containing an Arg-Gly-Asp (RGD) motif and calcium-binding EGF-like domains. It promotes adhesion of endothelial cells through interaction of integrins and the RGD motif. It is prominently expressed in developing arteries but less so in adult vessels. However, its expression is reinduced in balloon-injured vessels and atherosclerotic lesions, notably in intimal vascular smooth muscle cells and endothelial cells. Therefore, the protein encoded by this gene may play a role in vascular development and remodeling. Defects in this gene are a cause of autosomal dominant cutis laxa, autosomal recessive cutis laxa type I (CL type I), and age-related macular degeneration type 3 (ARMD3). |
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